Beta 2-agonist fenoterol has greater effects on contractile function of rat skeletal muscles than clenbuterol.

نویسندگان

  • James G Ryall
  • Paul Gregorevic
  • David R Plant
  • Martin N Sillence
  • Gordon S Lynch
چکیده

Potential treatments for skeletal muscle wasting and weakness ideally possess both anabolic and ergogenic properties. Although the beta(2)-adrenoceptor agonist clenbuterol has well-characterized effects on skeletal muscle, less is known about the therapeutic potential of the related beta(2)-adrenoceptor agonist fenoterol. We administered an equimolar dose of either clenbuterol or fenoterol to rats for 4 wk to compare their effects on skeletal muscle and tested the hypothesis that fenoterol would produce more powerful anabolic and ergogenic effects. Clenbuterol treatment increased fiber cross-sectional area (CSA) by 6% and maximal isometric force (P(o)) by 20% in extensor digitorum longus (EDL) muscles, whereas fiber CSA in soleus muscles decreased by 3% and P(o) was unchanged, compared with untreated controls. In the EDL muscles, fenoterol treatment increased fiber CSA by 20% and increased P(o) by 12% above values achieved after clenbuterol treatment. Soleus muscles of fenoterol-treated rats exhibited a 13% increase in fiber CSA and a 17% increase in P(o) above that of clenbuterol-treated rats. These data indicate that fenoterol has greater effects on the functional properties of rat skeletal muscles than clenbuterol.

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2-Agonist fenoterol has greater effects on contractile function of rat skeletal muscles than clenbuterol

Ryall, James G., Paul Gregorevic, David R. Plant, Martin N. Sillence, and Gordon S. Lynch. 2-Agonist fenoterol has greater effects on contractile function of rat skeletal muscles than clenbuterol. Am J Physiol Regul Integr Comp Physiol 283: R1386–R1394, 2002. First published September 5, 2002; 10.1152/ajpregu.00324.2002.—Potential treatments for skeletal muscle wasting and weakness ideally poss...

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عنوان ژورنال:
  • American journal of physiology. Regulatory, integrative and comparative physiology

دوره 283 6  شماره 

صفحات  -

تاریخ انتشار 2002